jethro
Distinguished member
- Joined
- Nov 2, 2017
- Messages
- 457
- Reason
- PALS
- Diagnosis
- 09/2017
- Country
- HR
- State
- Croatia
- City
- ZAGREB
you saw that movie, but i need (maybe new) discussion.
sci. articles said (just once!) that there is very often that pals have fatty liver/nafl. one trial said app 76%.
no direct connection with ALS and liver by now. But, when i had car accident, in 1997, the doctor said that i have alt and ast elevated. nothing critical (app. 2x upper limit of refference value), but i should check it at internist. so i was...
checked for viruses, willson's disease, all hepatitis - a, b, c...xyz. everything negative.
i didnt have any issues at all, but i checked it in 23 years for (maybe) 20 times. value was allways the same - elevated. +- 10%
if fatty liver/ast&alt is/are present permanent for a long term, it may trigger some processes, even genes alteration. hence, it is normally that nothing can be find in a examinations for a short time. epigenetic is on a stage! we dont need to emphaisze that healthy liver is a prerequisite against hyperammonia, which is considered as a possible cause of als.
currently, i have normal alt and ast (alt<ast!) thanks to cbd (cb2 receptors are in a liver!) for a year. still have faith in a reverse epigenetics, even i can not count zeros. (p=0.00000000000001%).
how is/was your liver?
thoughts?
sci. articles said (just once!) that there is very often that pals have fatty liver/nafl. one trial said app 76%.
no direct connection with ALS and liver by now. But, when i had car accident, in 1997, the doctor said that i have alt and ast elevated. nothing critical (app. 2x upper limit of refference value), but i should check it at internist. so i was...
checked for viruses, willson's disease, all hepatitis - a, b, c...xyz. everything negative.
i didnt have any issues at all, but i checked it in 23 years for (maybe) 20 times. value was allways the same - elevated. +- 10%
if fatty liver/ast&alt is/are present permanent for a long term, it may trigger some processes, even genes alteration. hence, it is normally that nothing can be find in a examinations for a short time. epigenetic is on a stage! we dont need to emphaisze that healthy liver is a prerequisite against hyperammonia, which is considered as a possible cause of als.
currently, i have normal alt and ast (alt<ast!) thanks to cbd (cb2 receptors are in a liver!) for a year. still have faith in a reverse epigenetics, even i can not count zeros. (p=0.00000000000001%).
how is/was your liver?
thoughts?