Janika1978
New member
- Joined
- Oct 11, 2019
- Messages
- 2
- Reason
- Learn about ALS
- Diagnosis
- 00/0000
- Country
- HU
- State
- HU
- City
- Sajóivánka
Hello!
Please help! Is this Als? My mother is unfortunately ill and very afraid! Thank you in advance for your help!
09/17/2019. ENG-EMG examination:
During the ENG examination, the jo. n. medianus and bo. n. peroneus stimulation only
low amplitude motor response can be recorded from proximal muscle.
The motor amplitudes of the other nerve examined, the jo. n. peroneus motoros
driving speeds have also decreased. Sensory parameters of the examined nerves
They held.
EMG examination:
All muscles have marked denervative activity (fibrillation
and fasciculatory potentials, positive sharp waves). The good. m.
except for sternocelidomastoideus, there is marked unit reduction in all muscles
visible. The average of the amplitudes of the motor unit potentials is jo. m.
sternocleidomastoideusban, his. m. vastus laterally and jo. m. the deltoid
rose, the good. m. parathoracalis in VI, bo. m. In I. dorsalis interosseus
and bo. m. tibialis in ant is normal. Duration of motor unit potentials
average in all muscles is normal.
Opinion:
Electrophysiologically it is of severe, motor and axonal type
polyneuropathy, and on-going denervation in all muscles
there are signs of subacutaneous neurogenic damage.
The finding corresponds to advanced motor neuron disease.Epikrízis:
Recruitment was made for additional studies. Electrophysiological studies
motoneuron disease. To study pyramid pathway dysfunction, MEP
examination is not possible due to severely damaged periphery. The patient
following his / her information and written consent, lumbarly, without complications
is characterized by a slight increase in protein and isoelectricity
focusing on both serum and cerebrospinal fluid indicates very faint oligoclonality
pattern was guessable. A liquor control test is recommended at a later date.
Due to the above differences, there is further direction towards paraneoplasia and tumor research
we recommend examinations. In our department we have ASA, Letrox, Coverex AS, Rilutek and
Rosuvastatin was given. His blood pressure was between 120/80 mmHg and 160/90 mmHg. On this day
after prior consultation, with unchanged neurological status, relatives
we'll take her home. When he is dismissed, he is cardio-pulmonary compensated, feverless,
there is no evidence of deep thrombosis.
Opinion:
Hypertension. - Infarctus lacunaris multiplex cerebri. Spondylosis et polydiscopathy
thoracolumbar. - Coxarthrosis incipiens lateris utriusque. - Protrusio disci LII et
LV. - Hypothyroidism. - Thyroiditis chronica. Motoneuron disease (primary muscularis
form of atrophia).
Please help! Is this Als? My mother is unfortunately ill and very afraid! Thank you in advance for your help!
09/17/2019. ENG-EMG examination:
During the ENG examination, the jo. n. medianus and bo. n. peroneus stimulation only
low amplitude motor response can be recorded from proximal muscle.
The motor amplitudes of the other nerve examined, the jo. n. peroneus motoros
driving speeds have also decreased. Sensory parameters of the examined nerves
They held.
EMG examination:
All muscles have marked denervative activity (fibrillation
and fasciculatory potentials, positive sharp waves). The good. m.
except for sternocelidomastoideus, there is marked unit reduction in all muscles
visible. The average of the amplitudes of the motor unit potentials is jo. m.
sternocleidomastoideusban, his. m. vastus laterally and jo. m. the deltoid
rose, the good. m. parathoracalis in VI, bo. m. In I. dorsalis interosseus
and bo. m. tibialis in ant is normal. Duration of motor unit potentials
average in all muscles is normal.
Opinion:
Electrophysiologically it is of severe, motor and axonal type
polyneuropathy, and on-going denervation in all muscles
there are signs of subacutaneous neurogenic damage.
The finding corresponds to advanced motor neuron disease.Epikrízis:
Recruitment was made for additional studies. Electrophysiological studies
motoneuron disease. To study pyramid pathway dysfunction, MEP
examination is not possible due to severely damaged periphery. The patient
following his / her information and written consent, lumbarly, without complications
is characterized by a slight increase in protein and isoelectricity
focusing on both serum and cerebrospinal fluid indicates very faint oligoclonality
pattern was guessable. A liquor control test is recommended at a later date.
Due to the above differences, there is further direction towards paraneoplasia and tumor research
we recommend examinations. In our department we have ASA, Letrox, Coverex AS, Rilutek and
Rosuvastatin was given. His blood pressure was between 120/80 mmHg and 160/90 mmHg. On this day
after prior consultation, with unchanged neurological status, relatives
we'll take her home. When he is dismissed, he is cardio-pulmonary compensated, feverless,
there is no evidence of deep thrombosis.
Opinion:
Hypertension. - Infarctus lacunaris multiplex cerebri. Spondylosis et polydiscopathy
thoracolumbar. - Coxarthrosis incipiens lateris utriusque. - Protrusio disci LII et
LV. - Hypothyroidism. - Thyroiditis chronica. Motoneuron disease (primary muscularis
form of atrophia).
