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Following is my own translation of the ENMG report from Aug 6, 2012:

NCV:
In the upper limbs, motoric responses were overall exceptionally low. Conduction velocities were within reference values with the exception of the left medianus, where the motoric conduction velocity on the wrist-elbow segment was exceptionally low. Distal motoric latencies in the upper limbs in both the median and ulnar nerve were exceptionally long. Latencies of the F responses were strongly prolonged with respect to the height and age of the patient.

F response could not be obtained from the left medianus. Sensory conduction velocities and responses in both upper limbs were normal. In the lower limbs, motoric and sensory conduction velocities and responses as well as latencies of the spinal reflections were normal throughout the line.

Needle EMG:
Muscles were examined in each limb, on the torso and in the bulbar region. The muscles in the lower limbs, torso and the bulbar region were intact. In the upper limbs, bilaterally in the muscles of the myotomic region of the upper, middle and lower branch of the brachial plexus, we note changes in line with long-lasting neurogenic damage, with fibrillations, fasciculation potentials, motor unit loss and polyphasic, reinnervated long-lasting unit potentials. In the left IDI muscle, no activating unit potentials were found, only spontane activity.

Conclusion:
The finding is in line with a high-degree chronic axonal neuropathy of the upper limbs. This kind of finding could be caused by a spinal level process, e.g. spinal muscular atrophy. Neurophysiologically the finding was limited to the upper limbs, so it would not be quite typical regarding the suggested ALS disease.
 
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