I ask of your forgiveness ahead of time if I am being overly apprehensive and cause any disrespect. We were told in a former post that some on here are great at the reading of EMG, NCV and SFEMG and translating them into layman's terms. I humbly and nervously am asking that of you, please. Nerves are from not wanting to be a nuisance.
"Nerve conductivity studies were performed on the left ulnar motor and sensory responses. There were no sensory response that could be recorded; however the motor response appeared to be fairly intact. Repetitive stimulation was performed on the left ulnar nerve and on the spinal accessory nerve on the left and both of these were normal and showed no appreciable electrode decrement before or after exercise"
"Concentric needle EMG was performed on the selected muscles in the left arm. There is a mild increase in motor unit amplitude and duration and the ulnar innervated first dorsal interosseous and also slight increase in amplitude and duration of the motor units in the median innervated flexor pollicis longus, which also showed some mildly decreased recruitment. Otherwise deltoid, triceps and EDC were normal."
Electrodiagnostic Interpretation: "This is an abnormal study showing a non-localizable ulnar neuropathy, which is consistent with his reported history of ulnar neuropathy. There is no evidence for myasthenia gravis on the repetitive stimulation test, although this is an insensitive test given his lack of symptoms in the peripheral muscles. The EMG portion did show signs of some mild denervation/reinnervation in the ulnar innervated first dorsal interosseous, which fits with his history of ulnar neuropthy; however, we also found mild denervation changes in the median innervated flexor pollicis longus, which is of uncertain clinical significance but these two findings together could suggest a mild C8 radiculopathy. Early motor neuron disease cannot be ruled out"
"Single Fiber EMG was done from the left frontalis muscle. The mean jiter was 41.76 microseconds which is above the ULN of 35.5. Four of 25 pairs had a jitter exceeding the ULN for single pairs. Two of these showed blocking.
Conclusion: "This was an abnormal study. There is an evidence of neuromuscular junction defect c/w myasthenia gravis although other neuromuscular diseases could produce similar findings"
Hate this is long and thank you in advance for reading and offering any knowledge on it.
"Nerve conductivity studies were performed on the left ulnar motor and sensory responses. There were no sensory response that could be recorded; however the motor response appeared to be fairly intact. Repetitive stimulation was performed on the left ulnar nerve and on the spinal accessory nerve on the left and both of these were normal and showed no appreciable electrode decrement before or after exercise"
"Concentric needle EMG was performed on the selected muscles in the left arm. There is a mild increase in motor unit amplitude and duration and the ulnar innervated first dorsal interosseous and also slight increase in amplitude and duration of the motor units in the median innervated flexor pollicis longus, which also showed some mildly decreased recruitment. Otherwise deltoid, triceps and EDC were normal."
Electrodiagnostic Interpretation: "This is an abnormal study showing a non-localizable ulnar neuropathy, which is consistent with his reported history of ulnar neuropathy. There is no evidence for myasthenia gravis on the repetitive stimulation test, although this is an insensitive test given his lack of symptoms in the peripheral muscles. The EMG portion did show signs of some mild denervation/reinnervation in the ulnar innervated first dorsal interosseous, which fits with his history of ulnar neuropthy; however, we also found mild denervation changes in the median innervated flexor pollicis longus, which is of uncertain clinical significance but these two findings together could suggest a mild C8 radiculopathy. Early motor neuron disease cannot be ruled out"
"Single Fiber EMG was done from the left frontalis muscle. The mean jiter was 41.76 microseconds which is above the ULN of 35.5. Four of 25 pairs had a jitter exceeding the ULN for single pairs. Two of these showed blocking.
Conclusion: "This was an abnormal study. There is an evidence of neuromuscular junction defect c/w myasthenia gravis although other neuromuscular diseases could produce similar findings"
Hate this is long and thank you in advance for reading and offering any knowledge on it.
